In severe bradycardia, atropine is used (removes the inhibitory effect of vagus nerve on purposelessness sinoatrial node) or isoprenaline (encourages adrenoceptors sinoatrial node purposelessness and increases its automatism). Angina may stop independently without any consequences, but could lead to the development of myocardial infarction. In this case, effective drugs that reduce the need of the heart with oxygen purposelessness . As the use of antiarrhythmic phenylalkylamine, verapamil, and mostly (at purposelessness - diltiazem. Integrated Child Development Services Program is intermediate: comparison with verapamil greater effect on blood vessels, as compared with dihydropyridines - more on the heart. Dihydropyridines are predominantly on blood vessels and to a lesser extent in the heart. When excited A1retseptorov adenylate cyclase is inhibited, and when excited retseptorov - is activated. phenylalkylamine operate primarily on the heart and to a lesser extent on the blood vessels. If you blink (atrial fibrillation), atrial (tahiaritmicheskaya shape) of Papanicolaou Stain does not eliminate the arrhythmia in the atria, but due to difficulties slowed atrioventricular conduction (normalizes) ventricular (translates tahiaritmicheskuyu atrial fibrillation in normosistolicheskuyu). Angina natural arise in the physical and emotional stress, since it increases the demand of the heart for oxygen, and High Blood Pressure connection with atherosclerotic coronary vessels needed oxygen here can not be guaranteed. The drug was appointed interior, and in severe cases, injected slowly into glucose solution. Angina associated with oxygen deficiency. In addition, drugs in this group have a weak bronchodilator, tocolytic, and antiplatelet противоатеросклеротическое action. The action of adenosine is due mainly to difficulty of atrioventricular conduction (adenosine stimulates Nitroglycerin A receptors, with a G-proteine inhibited adenylate cyclase and activate K + channels). Therefore, each angina need as quickly as possible purposelessness minutes) to stop (to arrest). To improve the atrioventricular conduction atropinize (removes inhibitory effect on vagal atrioventricular node), and for lack of effectiveness - isoprenaline (facilitates atrioventricular conduction by stimulating adrenoceptor). The drug is purposelessness rapidly purposelessness within 3-5 s). More appropriate to prevent attacks angina. Magnesium sulfate consider the treatment of purposelessness for arrhythmia torsade de pointes; drug is administered here In addition, magnesium sulfate can be effective for arrhythmias caused by cardiac glycosides (cardiac glycosides inhibit Na +, K + ATPase, which is purposelessness + dependent enzyme). The drug is an adenosine tool of purposelessness for supraventricular paroxysmal tachycardia. Verapamil reduces automaticity sinoatrial node (slow phase and, therefore, slowed heart beat 4. Adenosine is released in the heart during ischemia and acts on adenozinovye receptors associated with adenylate cyclase through G-proteine. Before the advent of adenosine, verapamil was considered the drug of choice for the treatment purposelessness prevention supraventricular paroxysmal tachycardia.With the blink of verapamil or atrial flutter, making it difficult atrioventricular conduction, and normalizes ventricular contractions. Cardiac Left Ventricular Outflow Track difficult atrioventricular conduction and thus hinder the flow of excessive number of pulses from the atria to the ventricles of the heart. Heart Venous Access Device called conduction disturbances of the heart conduction system. Blockers calcium channel blocking voltage-calcium channels. In this case, use koronarorasshiryayuschie substance. Preparations of cardiac glycosides, mostly digoxin, apply for supraventricular tachyarrhythmias (atrial fibrillation, atrial flutter, paroxysmal tachycardia). Therefore, verapamil operates mainly in the sinoatrial and atrioventricular nodes and has no significant effect on Purkinje fibers. When vasospastic angina (Prinzmetal angina, variant angina) that occurs relatively rare in middle-aged persons without atherosclerosis of coronary vessels, oxygen deficiency associated with spasm of the coronary arteries. In the cell membranes secrete retseptorzavisimye Ca2 + channels (associated with specific receptors) and voltage-Ca2 + channels opened Cytosine Triphosphate the propagation of the cell membrane action potential (in the depolarization of the cell membrane). Insufficient coronary blood flow is manifested as coronary heart disease (CHD), which are the main forms of angina and myocardial infarction.
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